https://www.researchgate.net/publication/267744684_Review_of_Toluene_Actions_Clinical_Evidence_Animal_Studies_and_Molecular_Targets
>Inhaled toluene acts as a robust discriminative stimulus [85,86] and also produces CNS depressant-like [15,45,77], amphetamine-like [14], and PCP-like discriminative effects [25]. Similar to other CNS depressant drugs, toluene has anxiolytic-like propertis [24,63,74], anticonvulsant effects [26,29,31,35,96], and impairs locomotor coordination [28,89]. It also exerts antidepressant-like actions [39] and a biphasic locomotor response; that is, increased and decreased activity at low and high concentrations, respectively
>toluene was shown to significantly inhibit the NMDA subtype of glutamate-activated ion channels while having little effect on the closely-related AMPA subtype of ionotropic glutamate receptors
>Moreover, within the NMDA family, receptors composed of the GluN2B subunit were considerably more sensitive to toluene inhibition than other NMDA receptor subtypes. A similar effect was observed for nicotinic acetylcholine receptors (nAchRs), where α4β2 receptors were much more sensitive to toluene inhibition than α7 nAchRs [5]. Amongst the P2X family of ATP-gated channels, toluene inhibits the function of some subtypes (P2X2, P2X4) but enhances currents through P2X3 containing receptors
>Other researchers have found that toluene produces increases in dopamine release and dopaminergic neurons’ activity [10,47,79,80,97], regional brain changes in glutamate, glutamine, and monoamine levels [7,76,95], as well as changes in NMDA and GABAA receptor densities or subunit composition

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